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The similarly-sized associations between increased FGF-23 concentration and cardiovascular (atherosclerotic and nonatherosclerotic) and noncardiovascular outcomes, together with the absence of any exposure-response relationship, suggest that the relationship between FGF-23 and cardiovascular disease risk may be noncausal.

Original publication

DOI

10.1681/asn.2017121334

Type

Journal article

Journal

Journal of the american society of nephrology : jasn

Publication Date

15/05/2018

Addresses

Health Economics Research Centre.